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Dermatologic Diseases - Statistics
General Stats
- This quiz has been taken 1 time
- The average score is 33 of 185
Answer Stats
| Hint | Answer | % Correct |
|---|---|---|
| CAUSE: Cellulitis | CAUSE: Acute bacterial infx of dermis and subcutaneous tissues, often group A strep or staph aureus (except in immunocompromised). Commonly lower extremity. Bacteria inoculated thru skin, release virulence factors/toxins, activate immune system | 100%
|
| CAUSE: Delusions of Parasitosis | CAUSE: False delusion of parasite infections | 100%
|
| CAUSE: Verruca Vulgaris (common wart) | CAUSE: HPV infection - enters thru broken skin, infects basal keratinocytes, replicates w keratinocyte/vascular proliferation, evades immune system by preventing MHC antigenic expression. | 100%
|
| CAUSE: Contact Dermatitis | CAUSE: Irritant (chemical/mechanical irritation directly damages skin) vs. allergic (allergen causes type IV delayed hypersensitivity). Often poison ivy. | 100%
|
| CAUSE: Urticaria (Hives) | CAUSE: Mast cell degranulation triggered by drugs, foods, infx, stress, autoimmune = mast cell degranulation = histamine release, capillary permeability | 100%
|
| CAUSE: Varicella zoster virus (shingles) | CAUSE: Reactivation of primary varicella (chickenpox) | 100%
|
| DX: Scabies | DX: Clinical, skin scraping (mite or feces) | 100%
|
| DX: Liver disease / Cirrhosis | DX: Liver function tests (AST/ALT) | 100%
|
| DX: Herpes simplex virus | DX: Viral PCR | 100%
|
| INTERLEUKINS: Psoriasis | INTERLEUKINS: IL-17, IL-23 | 100%
|
| PRESENTATION: Melasma | PRESENTATION: Acquired hyperpigmentation of the skin typically affecting sun-exposed areas of the face, more commonly in child-bearing women and darker skin types. | 100%
|
| PRESENTATION: Erythema Chronicum Migrans (Lyme) | PRESENTATION: Begins as small red macule, spreads to large targetoid patch. May have fevers, chills. | 100%
|
| PRESENTATION: Varicella zoster virus (shingles) | PRESENTATION: Blistering lesions resembling HSV in dermatomal distribution. Disseminated disease (across dermatomes) highly infectious, requires hospitalization | 100%
|
| PRESENTATION: Kawasaki's Disease | PRESENTATION: Fever lasting over 5 days. Strawberry tongue (extreme erythema of tongue), bilateral conjunctivitis, swollen/red hands, erythematous desquamating patches on extremities/buttocks, cervical lymphadenopathy. May induce vasculitis with CV complications (aneurysms, MI). | 100%
|
| PRESENTATION: Alopecia areata | PRESENTATION: Hair falls out in small circles, progresses to entire scalp then body | 100%
|
| PRESENTATION: Pityriasis Rosea | PRESENTATION: Herald lesion (largest and first-appearing), spreads to trunk in "christmas tree pattern" w colarette of scale | 100%
|
| PRESENTATION: Tinea versicolor | PRESENTATION: Hypopigmented-to-hyperpigmented well-demarcated macules coalescing into patches, SCALY WHEN STRETCHED (furfuricious scale). Multiple color patterns (tan-pink, hypopigmented in dark skin). Chest, back, upper arms, abdomen, thighs. Generally asymptomatic | 100%
|
| PRESENTATION: Scabies | PRESENTATION: Itchy rash in webspaces of hands/feet, wrists, armpits, navel, groin. In critically ill or immunocompromised, may be crusted. | 100%
|
| PRESENTATION: Molluscum contagiosum | PRESENTATION: Resembles warts. Single-to-multiple, yellow or pink, shiny, umbilicated papules. May itch. | 100%
|
| PRESENTATION: Keloid | PRESENTATION: Smooth overgrown scar extending past boundary of injury. Can be painful or itchy. | 100%
|
| PRESENTATION: Psoriasis | PRESENTATION: Well-circumscribed erythematous plaques with overlying silver-white scale at the elbows, knees, scalp, lumbosacral region. Classically not itchy. Koebner phenomenon (occurs at sites of injury/irritation). Several types: plaque, guttate (scattered red papules scale), inverse (occurs in body folds w no scale). NO MACULES ON HANDS OR FEET (unlike secondary syphilis). Comorbid w metabolic syndrome, diabetes, stroke, MI/CVA, psoriatic arthritis (dactylitis). | 100%
|
| TX: Urticaria (Hives) | TX: Acute (antihistamines, topical steroids) vs. chronic (H1 antihistamines + H2 antihistamines/leukotriene modifiers + steroids + immunosuppressants) | 100%
|
| TX: Verruca Vulgaris (common wart) | TX: Destructive (cryotherapy, laser, salicylic acid), immunologic (Imiquimod, Candida Ag, Cimetidine, vaccination) | 100%
|
| TX: Delusions of Parasitosis | TX: Difficult to tx, as patients usually unwilling to see psychiatrist. Tx underlying disease (thyroid, vitamin B12), topical moisturizers/steroids, doxepin, atypical antipsychotics | 100%
|
| TX: Liver disease / Cirrhosis | TX: Evaluate liver disease, manage itching (topical moisturizers/corticosteroids, cholestyramine, narrow band UV) | 100%
|
| TX: Scabies | TX: Permethrin cream, Ivermectin, tx close contacts | 100%
|
| TX: Acne | TX: Topicals (benzoyl peroxide, saliclyic acid, antibiotics, retinoids), systemic (antibiotics, OCPs, spironolactone, isotretinoin) | 100%
|
| TX: Seborrheic Dermatitis (Dandruff) | TX: Tx yeast (selenium sulfide shampoos, ketoconazole), tx scaling (salicylic acid) topical steroids, topical calcineurin inhibitors, Apremilast (PDE-4 inhibitor) | 100%
|
| TYPE: Molluscum contagiosum | TYPE: Infectious | 100%
|
| TYPE: Varicella zoster virus (shingles) | TYPE: Infectious | 100%
|
| TYPE: Seborrheic Dermatitis (Dandruff) | TYPE: Inflammatory | 100%
|
| TYPE: Urticaria (Hives) | TYPE: Inflammatory | 100%
|
| TYPE: Acne | TYPE: Inflammatory | 100%
|
| CAUSE: Vitiligo | CAUSE: Acquired autoimmune disorder of pigmentation w loss of epidermal melanocytes resulting in depigmented patches. Genetic predisposition activated by environmental trigger/stress, autoimmune T cell process acting on JAK/STAT = kills melanocytes. | 0%
|
| CAUSE: Melasma | CAUSE: Acquired hyperpigmentation of the skin resulting from chronic UV exposure (increases MMP2/9 = decrades collagen, melanin descends into dermis). Female hormonal stimulation, genetics involved. | 0%
|
| CAUSE: Acne | CAUSE: Androgen sensitivity of pilosebaceous unit = obstructive plugs = follicle wall rupture = inflammation = infection, sebum (?). Triggered by stress, occlusion, medications. | 0%
|
| CAUSE: Dermatomyositis | CAUSE: Assoc w malignancy (preceding, co-occurring, following), mostly within first year w elevated risk for 3-5 years. Ovarian cancer overrepresented. Also w interstitial lung disease & aspiration due to esophageal involvement | 0%
|
| CAUSE: Pyoderma Gangrenosum | CAUSE: Assoc w underlying disease (IBD, hematologic malignancy) | 0%
|
| CAUSE: Bullous pemphigoid | CAUSE: Autoantibodies to BP180 and BP230 of hemidesmosomes disrupt epidermal attachemnt to TEJ, causing blistering/erosion, severe pruritis. | 0%
|
| CAUSE: Pemphigus vulgaris | CAUSE: Autoantibodies to DSG-1 and DSG-3 in desmosomes (connect keratinocytes), causing blistering. | 0%
|
| CAUSE: Alopecia areata | CAUSE: Autoimmune hair loss | 0%
|
| CAUSE: Grave's Disease | CAUSE: Autoimmune hyperthyroidism. | 0%
|
| CAUSE: Seborrheic Dermatitis (Dandruff) | CAUSE: Chronic inflammatory disorder resulting from skin barrier dysfx and immne rxn to Malssezia yeast. | 0%
|
| CAUSE: Melanocytic Nevus (mole) | CAUSE: Conglomerations of melanocytes (derived from neural crest), most often in sun-exposed areas | 0%
|
| CAUSE: DRESS Syndrome | CAUSE: Drug rxn w eosinophilia and systemic sx. Systemic immune response triggered by longer term drug exposure reactivation of underlying latent virus. Many causative drugs, especially aromatic anticonvulsants (i.e. carbamazepine) | 0%
|
| CAUSE: Tinea Corporis (Ringworm) | CAUSE: Fungal infx of epidermus and stratum corneum by dermatophytes (Trychophyton/Microsporum). Enter thru direct contact w broken skin, produce keratinases (digest keratin) = inflammatory response = redness, spreads outward from origin | 0%
|
| CAUSE: Hidradenitis Suppurativa | CAUSE: Genetic factors (hair follicle genes), elevated cytokines, environmental factors, hormonal factors lead to inflammatory nodule formation in skin folds. | 0%
|
| CAUSE: Psoriasis | CAUSE: Genetic predisposition with environmental trigger activates inflammatory pathway involving TH17, IL-17. | 0%
|
| CAUSE: Telogen effluvium | CAUSE: Hairs shift into 'shed' phase, stress-related hair loss | 0%
|
| CAUSE: Solar/Simple Lentigo | CAUSE: Hyperpigmentation of keratinocytes due to sun damage. NOT a melanocytic lesion - autophagy of keratinocytes to neighboring melanocyte. | 0%
|
| CAUSE: Lichen Planus | CAUSE: Inflammatory dermatitis in which T cells react to unknown antigen at dermal-epidermal junction. May be drug-induced or assoc w Hep C. | 0%
|
| CAUSE: Rosacea | CAUSE: Inflammatory disorder related to demodex mite, triggered by sun, spicy food, wine, steroids | 0%
|
| CAUSE: Keloid | CAUSE: Injury leads to upregulated cytokines, fibroblast proliferation, and ECM synthesis | 0%
|
| CAUSE: Liver disease / Cirrhosis | CAUSE: Liver disease (alcoholic/viral/autoimmune hepatitis, malignancy) | 0%
|
| CAUSE: Scabies | CAUSE: Mite sarcoptes scabiei, person-to-person contact, very common. | 0%
|
| CAUSE: Molluscum contagiosum | CAUSE: Molluscipox virus, skin-to-skin contact or STI | 0%
|
| CAUSE: Herpes simplex virus | CAUSE: Neurtropic dsDNA virus. Virally-mediated destruction of keratinocytes forms blisters | 0%
|
| CAUSE: Pyoderma gangrenosum | CAUSE: Neutrophilic infiltration causes soft tissue necrosis resembling an infx. | 0%
|
| CAUSE: Erythema nodosum | CAUSE: Panniculitis (fat inflammation) | 0%
|
| CAUSE: Coma Bullae | CAUSE: Pressure-induced keratinocyte necrosis, within 48-72 hrs of imobility. Clamps off blood vessels = skin cells die = blister forms. | 0%
|
| CAUSE: Acanthosis Nigricans | CAUSE: Proliferative disorder resulting from insulin resistant states (obesity, diabetes mellitus) = epidermal growth and thickening | 0%
|
| CAUSE: Stevens-Johnson Syndrome or Toxic Epidermal Necrolysis | CAUSE: Severe T-cell-mediated allergic rxn to systemic medication 7-21 days after drug initiation, often sulfonamides, anticonvulsants, allopurinol, lamotrigine. Full-thickness keratinocyte apoptosis and necrosis. | 0%
|
| CAUSE: Atopic Dermatitis (Eczema) | CAUSE: Skin barrier dysfx (genetic, mutations in fillagrin? itch-scratch cycle, exposure to environmental antigen), inflammatory dysregulation (skewed to Th2 cytokines). Part of atopic triad (allergic march) | 0%
|
| CAUSE: Bullous impetigo | CAUSE: Skin infx w staph aureus or group A strep. Exfoliative toxin causes blistering (cleaves desmosomes of upper epidermis) | 0%
|
| CAUSE: Stasis dermatitis | CAUSE: Swelling in legs (irritation/inflammation) | 0%
|
| CAUSE: Adrogenetic Alopecia | CAUSE: Testosterone (converted to DHT) miniaturizes and eliminates terminal hairs. Males over 40, post-menopausal females. | 0%
|
| CAUSE: Erythema Chronicum Migrans (Lyme) | CAUSE: Tickborne infection by spirochete, 7-14 days post-bite. | 0%
|
| CAUSE: Morbilliform Eruption | CAUSE: Type IV hypersensitivty rxn to drug or one of its metabolites. T-cell-mediated, triggers immune cascade. | 0%
|
| CAUSE: Kawasaki's Disease | CAUSE: Unknown, possibly viral | 0%
|
| CAUSE: Pityriasis Rosea | CAUSE: Viral exanthem (HHV-6) | 0%
|
| CAUSE: Scurvy | CAUSE: Vitamin C deficiency (alcohlism, gastric bypass, malabsoprtive syndromes). Cofactor in collagen production = weakens blood vessels, bones/teeth, skin/hair. | 0%
|
| CAUSE: Tinea versicolor | CAUSE: Yeast infx by Malassezia furfur (normal organism on skin) - lipophilic organism overgrows in oily skin/sweat, converts to yeast. Inhibits tyrosinase in melanin synthesis | 0%
|
| DX: Acanthosis Nigricans | DX: A1C, check family hx | 0%
|
| DX: Pemphigus vulgaris | DX: Biopsy (H&E and IF), DSG-1 and DSG-3 serologies | 0%
|
| DX: Pyoderma Gangrenosum | DX: Biopsy (shows neutrophilic infiltration) and tissue cultures (will be negative). Frequently misdiagnosed | 0%
|
| DX: Bullous pemphigoid | DX: Biospy (H&E and IF - subepidermal split and strong line at dermoepidermal junction), BP-180/230 serologies | 0%
|
| DX: Lichen Planus | DX: Clinical, biopsy, differentiate from psoriasis by purple color | 0%
|
| DX: Contact Dermatitis | DX: Clinical dx, history (occupation, exposures, hobbies), physical exam (exposure distribution), patch testing for allergen | 0%
|
| DX: Tinea versicolor | DX: Clinical, KOH ("spaghetti and meatballs" hyphae and spores) | 0%
|
| DX: Delusions of Parasitosis | DX: Dx of exclusion. Rule out all organic causes before concluding delusion. | 0%
|
| DX: Tinea Corporis (Ringworm) | DX: KOH prep, culture, biopsy | 0%
|
| DX: Erythema Chronicum Migrans (Lyme) | DX: Serologic test (ELISA, Western blot), test ticks for lyme | 0%
|
| DX: Bullous impetigo | DX: Swab blisters for bacterial cx | 0%
|
| DX: Grave's Disease | DX: TSH, free T4 | 0%
|
| DX: Varicella zoster virus (shingles) | DX: Viral PCR | 0%
|
| INTERLEUKINS: RA | INTERLEUKINS: TNF, IL-6, IL-1, RANKL | 0%
|
| PRESENTATION: Tinea Corporis (Ringworm) | PRESENTATION: Annular to round, scaly red - brown plaques w central clearing and leading scale. Athlete's foot (maceration/moist white skin breakdown bt toes, painful/itchy), tinea capitis (scaly red plaques w hair breakage, itchy), onychomycosis/tinea unguis (white-yellow discoloration w subungual debris and crumbling nails) | 0%
|
| PRESENTATION: Herpes simplex virus | PRESENTATION: Blistering virus, affecting oral or genital regions (cold sores, fever blisters) | 0%
|
| PRESENTATION: Solar/Simple Lentigo | PRESENTATION: Brown macules, usually in sun-exposed areas | 0%
|
| PRESENTATION: Adrogenetic Alopecia | PRESENTATION: Decreased hair density and/or miniaturization of frontal hairline, sometimes vertex/partline. Temporal/occipital scalp generally preserved. | 0%
|
| PRESENTATION: Vitiligo | PRESENTATION: Depigmented patches. Non-segmental vs. segmental (rapid onset, recalcitrant, dermatomal). Active disease marked by Koebner phenomenon (trauma-induced spreading), confetti-like morphology, trichrome coloration, inflammation (pink/inflamed appearance). | 0%
|
| PRESENTATION: Telogen effluvium | PRESENTATION: Diffuse, sudden hair loss following stressful event/period | 0%
|
| PRESENTATION: Stevens-Johnson Syndrome or Toxic Epidermal Necrolysis | PRESENTATION: Dusky tender patches w bullae and skin fragility/sloughing. Nikolsky (pushing on skin forms blister) and Asboe-Hansen (blister expands when pushed) sign positive. Prodrome of fever, malaise, arthralgias. Acute onset tender skin. SJS if under 10%, TEN if over 30% of surface area. Ocular, mucosal, genital involvement. | 0%
|
| PRESENTATION: Pemphigus vulgaris | PRESENTATION: Epidermal blisters on skin/mucous membranes. Painful, itchy, burning. Life-threatening if untreated. | 0%
|
| PRESENTATION: Rosacea | PRESENTATION: Erythrotelangiectatic, papulopustular, rhinophymatous, ocular | 0%
|
| PRESENTATION: Dermatomyositis | PRESENTATION: Heliotrope + Gottron's papules (flat topped, erythematous to violaceous somewhat scaly papules predominantly occurring over extensor surfaces of hand joints). Both pathognomonic. Skin rash plus muscle involvement (though many never have clinically significant muscle involvement). Nail fold capillary changes (pericuticular erythema, micro-infarcts). Photosensitive condition ("shawl sign"). Muscle weakness ranging from non-clinically impairment to debilitating | 0%
|
| PRESENTATION: Bullous impetigo | PRESENTATION: Honey-colored crust and flaccid, superficial, fragile blisters. May develop staphylococcal scalded skin syndrome when toxin enters blood, causing widespread superficial bullae/erosions (though cx negative bc no disseminated infx). | 0%
|
| PRESENTATION: Hidradenitis Suppurativa | PRESENTATION: Inflammatory nodules in folds of skin, more common in black individuals. Painful, draining, often foul odor. | 0%
|
| PRESENTATION: Delusions of Parasitosis | PRESENTATION: Linear excoriations, prurigo nodules, ulcerations, spares areas the patient cannot reach | 0%
|
| PRESENTATION: Pyoderma Gangrenosum | PRESENTATION: Nonhealing ulcer despite routine wound/infection healing management, often on lower extremities. Can show pathergy (worsens w trauma at site including biopsy). If mistaken for infx, can be debrided = ulcer worsens! | 0%
|
| PRESENTATION: Pyoderma gangrenosum | PRESENTATION: Non-healing ulcer often assoc w IBD and malignancy. Not responsive to Abx | 0%
|
| PRESENTATION: Coma Bullae | PRESENTATION: Non-inflamed bullae on dependent sites in immobilized patients. May be red/purple w hemorrhage | 0%
|
| PRESENTATION: Seborrheic Dermatitis (Dandruff) | PRESENTATION: Occurs in areas rich in sebaceous glands (scalp, eyebrows, nasal folds, retroauricular, presternal). May be itchy. Cradle cap in infants. May occur in assoc w psoriasis. Petaloid variant presents on scalp and can extend onto face. | 0%
|
| PRESENTATION: Acne | PRESENTATION: Open comedones (blackheads) vs. closed comedones (whiteheads, pustules). Cysticnodulocystic acne is severe and can have permanent scarring. May cause hyperpigmentation. | 0%
|
| PRESENTATION: Liver disease / Cirrhosis | PRESENTATION: Palmar erythema, Terry's nails (whitened nailbeds), spider angiomas/telangiectasis, pruritis, jaundice | 0%
|
| PRESENTATION: Scurvy | PRESENTATION: Petechiae and ecchymoses, perifollicular hemorrhage, corkscrew hairs, hemorrhagic gingivitis, halitosis, loose teeth | 0%
|
| PRESENTATION: Melanocytic Nevus (mole) | PRESENTATION: Pink-to-brown well-circumscribed macules or papules. Evaluate risk based on asymmetry, borders, colors, diameter, evolution | 0%
|
| PRESENTATION: Stasis dermatitis | PRESENTATION: Pitting edema, small purpuric non-blanching areas, signs of chronic venous stasis, thickened skin, itchy | 0%
|
| PRESENTATION: Atopic Dermatitis (Eczema) | PRESENTATION: Poorly circumscribed, erythematous patches w excoriations at flexural surfaces. Tends to improve with age. Lichenification, post-inflammatory changes (hypopigmentation, hyperpigmentation) | 0%
|
| PRESENTATION: Grave's Disease | PRESENTATION: Pretibial myxedema, opthalmopathy (bulging eyes), acropachy (bulging of proximal nail fold) | 0%
|
| PRESENTATION: Lichen Planus | PRESENTATION: PURPLE, polygonal, pruritic, papules affecting skin, mucosal surfaces, nails, hair. Pruritic. Can cause Wickham's striae. | 0%
|
| PRESENTATION: DRESS Syndrome | PRESENTATION: Rash (morbilliform), heme (eosinophilia, atypical lymphocytes), systemic sx (hepatitis, interstitial nephritis, lymphadenopathy, thyroiditis, carditis). 2 weeks - 3 months after starting medication. | 0%
|
| PRESENTATION: Contact Dermatitis | PRESENTATION: Sharp lines of demarcation, geometric/linear shapes, bullae and vesicles in severe rxns | 0%
|
| PRESENTATION: Erythema nodosum | PRESENTATION: Tender multifocal nodules on lower extremities | 0%
|
| PRESENTATION: Bullous pemphigoid | PRESENTATION: Tense blisters and erosions of skin/mucous memranes, often w urticarial or eczematous base. Severe pruritis. Tends to have eosinophilic infiltrate. | 0%
|
| PRESENTATION: Cellulitis | PRESENTATION: UNILATERAL erythema, edema, tenderness, warmth. Acute, painful, indurated/swollen, rarely increased WBC, fever, bullae. (Do not confuse for DVT, erythema nodosum, stasis dermatitis) | 0%
|
| PRESENTATION: Acanthosis Nigricans | PRESENTATION: Velvety plaques manifesting in body folds in patients w insulin resistance. Cutaneous signs of diabetes (diabetic dermopathy, xanthelasma, skin tags) | 0%
|
| PRESENTATION: Verruca Vulgaris (common wart) | PRESENTATION: Verrucous (thickened/hyperpoliferative skin), thromobsed capillaries (black dots), single or clustered or scattered, mucosal or nonmucosal. If extensive/refractory, consider immunosuppression. | 0%
|
| PRESENTATION: Urticaria (Hives) | PRESENTATION: Wheals, individually lasting under 24 hours. Very itchy. May be acute (<6 wks) or chronic (>6 wks). | 0%
|
| PRESENTATION: Morbilliform Eruption | PRESENTATION: Widespread erythematous macules and thin papules on trunk/extremities. 7-14 days after exposure. Starts on the chest and moves to extremities, patient looks well, typically asymptomatic or pruritic. Reexposure can cause rapid recurrence. | 0%
|
| TX: Bullous impetigo | TX: Abx, topical if localized, consider MRSA coverage | 0%
|
| TX: Cellulitis | TX: Appropriate Abx regimen, often outpatient. Caution for gas/crepitus, necrosis, rapid progression. | 0%
|
| TX: Kawasaki's Disease | TX: Aspirin for CV complications (monitor for Reye syndrome - brain/liver failure) | 0%
|
| TX: Contact Dermatitis | TX: Avoidance, barriers, topical steroids, oral steroids if severe, antihistamines for pruritis | 0%
|
| TX: Rosacea | TX: Avoid triggers, acne treatments | 0%
|
| TX: Pemphigus vulgaris | TX: B cell suppression (corticosteroids, rituximab, IVIG) | 0%
|
| TX: Melanocytic Nevus (mole) | TX: Benign, no tx necessary, but melanomas may develop from pre-existing nevus and biopsy may be needed in worrisome cases | 0%
|
| TX: Bullous pemphigoid | TX: Corticosteroids, oomalizumab, rituximab, steroid-sparing immunosuppressives | 0%
|
| TX: Erythema Chronicum Migrans (Lyme) | TX: Doxycycline | 0%
|
| TX: Acanthosis Nigricans | TX: Environmental modifications (diet, weight loss), for skin plaques use moisturizers w keratolytics (lactic acid, urea cream, salicylic acid), topical retinoids | 0%
|
| TX: Melasma | TX: Inhibit tyrosinase (decreases melanin production), sunscreen (prefer tinted sunscreens w iron oxides), flucinolone, hydroquinone, tretinoin. | 0%
|
| TX: Solar/Simple Lentigo | TX: None needed, can destroy w laser. Sunscreen prevention key. Monitor to rule out melanoma. | 0%
|
| TX: Vitiligo | TX: Nonsegmental - topical tx, UVB, surgical if no response, systemic oral steroids if rapid progression, opzelura | 0%
|
| TX: Molluscum contagiosum | TX: Nothing in children, cryotherapy/electrodessication in adults. Self-limiting but may last years. | 0%
|
| TX: Tinea Corporis (Ringworm) | TX: Prevention, keep dry and air-exposed, antifungal cream and/or powder (miconazole, clomitrazole), topical antifungals, systemic antifungals | 0%
|
| TX: Keloid | TX: Prevention (no traumatic procedures), corticosteroid injection, lasers, rarely radiation if large/nonresponsive/functionally impairing | 0%
|
| TX: Stasis dermatitis | TX: Remove fluid, compression stockings, topical corticosteroids | 0%
|
| TX: Erythema nodosum | TX: Remove trigger, NSAIDs | 0%
|
| TX: Coma Bullae | TX: Reposition patient, supportive care (drain fluid PRN) | 0%
|
| TX: Pityriasis Rosea | TX: Self-resolving in 6-12 weeks | 0%
|
| TX: Atopic Dermatitis (Eczema) | TX: Skin-directed (corticosteroids, topical calcineurin inhibitors, phototherapy), systemics (small molecules), targeted (dupilumab) | 0%
|
| TX: Psoriasis | TX: Skin-directed (corticosteroids, vitamin D3 analogs, phototherapy, etc.), systemics (small molecules, biologics for IL-17) | 0%
|
| TX: Lichen Planus | TX: Skin-directed (topical steroids, topical calcineuron inhibitors, phototherapy) vs. systemic (oral corticosteroids, methotrexate, azathiorpine) | 0%
|
| TX: Hidradenitis Suppurativa | TX: Staged using Hurley Stages I-III (isolated boils, recurring boils w scarring and sinus tracts, widespread boils w interconnected tracts). Antibiotics (topical -> systemic), escalates to biologics and surgical excision. Pain management, superinfection tx, weight loss. | 0%
|
| TX: Pyoderma gangerenosum | TX: Steroids. DO NOT DEBRIDE. Do culture just inc ase to ENSURE it isn't a deep infx before you give immunosuppressants | 0%
|
| TX: DRESS Syndrome | TX: Stop all drugs that could be responsible, trend organ dysfx (if worsens, add steroids), taper steroids to prevent flares. | 0%
|
| TX: Morbilliform Eruption | TX: Stop the drug unless necessary, rule out dangerous drug rashes, treat sx, oral antihistimanes & topical steroids for pruritis | 0%
|
| TX: Telogen effluvium | TX: Supportive (hairs regrow after stress ends), Minoxidil | 0%
|
| TX: Stevens-Johnson Syndrome or Toxic Epidermal Necrolysis | TX: Supportive tx (wound care, humectant application), manage fluid/electrolyte abnormalities, aggressive tx. Active therapies controversial. | 0%
|
| TX: Adrogenetic Alopecia | TX: Topical (minoxidil, finasteride, DHT blockers), systemic (finasteride, spironolactone, minoxidil), procedural (hair transplant) | 0%
|
| TX: Tinea versicolor | TX: Topical (selenium sulfide shampoo, azoles), systemic (azoles) | 0%
|
| TX: Grave's Disease | TX: Tx hyperthyroidism, topical steroids/moisturizers, compression stockings | 0%
|
| TX: Herpes simplex virus | TX: Valacyclovir | 0%
|
| TX: Varicella zoster virus (shingles) | TX: Valacyclovir | 0%
|
| TX: Scurvy | TX: Vitamin C | 0%
|
| TYPE: Pemphigus vulgaris | TYPE: Immunologic | 0%
|
| TYPE: Bullous pemphigoid | TYPE: Immunologic | 0%
|
| TYPE: Stevens-Johnson Syndrome or Toxic Epidermal Necrolysis | TYPE: Immunologic | 0%
|
| TYPE: Vitiligo | TYPE: Immunologic | 0%
|
| TYPE: Morbilliform Eruption | TYPE: Immunologic | 0%
|
| TYPE: DRESS Syndrome | TYPE: Immunologic | 0%
|
| TYPE: Verruca Vulgaris (common wart) | TYPE: Infectious | 0%
|
| TYPE: Tinea Corporis (Ringworm) | TYPE: Infectious | 0%
|
| TYPE: Tinea versicolor | TYPE: Infectious | 0%
|
| TYPE: Pityriasis Rosea | TYPE: Infectious | 0%
|
| TYPE: Cellulitis | TYPE: Infectious | 0%
|
| TYPE: Erythema Chronicum Migrans (Lyme) | TYPE: Infectious | 0%
|
| TYPE: Scabies | TYPE: Infectious | 0%
|
| TYPE: Herpes simplex virus | TYPE: Infectious | 0%
|
| TYPE: Bullous impetigo | TYPE: Infectious | 0%
|
| TYPE: Hidradenitis Suppurativa | TYPE: Inflammatory | 0%
|
| TYPE: Psoriasis | TYPE: Inflammatory | 0%
|
| TYPE: Atopic Dermatitis (Eczema) | TYPE: Inflammatory | 0%
|
| TYPE: Contact Dermatitis | TYPE: Inflammatory | 0%
|
| TYPE: Lichen Planus | TYPE: Inflammatory | 0%
|
| TYPE: Coma Bullae | TYPE: Mechanical | 0%
|
| TYPE: Adrogenetic Alopecia | TYPE: Non-infectious | 0%
|
| TYPE: Keloid | TYPE: Skin growth | 0%
|
| TYPE: Melanocytic Nevus (mole) | TYPE: Skin growth | 0%
|
| TYPE: Delusions of Parasitosis | TYPE: Systemic | 0%
|
| TYPE: Scurvy | TYPE: Systemic | 0%
|
| TYPE: Kawasaki's Disease | TYPE: Systemic | 0%
|
| TYPE: Liver disease / Cirrhosis | TYPE: Systemic | 0%
|
| TYPE: Grave's Disease | TYPE: Systemic | 0%
|
| TYPE: Acanthosis Nigricans | TYPE: Systemic | 0%
|
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