cardiovascular drugs - Statistics

General Stats
  • This quiz has been taken 25 times
  • The average score is 23 of 68
Answer Stats
Hint Answer % Correct
Beta blockers
68%
What are the positive inotropes? Digoxin
63%
Digoxin
63%
What are the negative chronotropes? Digoxin
63%
Calcium channel blockers
58%
calcium channel blockers
58%
What are the negative inotropes? calcium channel blockers
58%
calcium channel blockers
58%
What are the vasodilators? Angiotensin Converting Enzyme (ACE) inhibitors
58%
What are examples of each? Perindopril (and other -prils)
58%
Dobutamine
53%
Amiodarone
47%
Atenolol
47%
Atenolol
47%
Atenolol
47%
Irbesartan (and other -sartans)
47%
What are the diuretics? loop diuretics (renal diuretic)
47%
Nitrates
47%
potassium channel blockers
47%
potassium sparing diuretics (renal diuretic)
47%
What type of blocker is each? sodium channel blockers
47%
used on occasion in preference to frusemide if concerned about potassium levels
47%
Amlodipine
42%
What are examples of these? Amlodipine
42%
cerebral oedema (after head trauma)
42%
What are examples of these? Frusemide
42%
Mannitol
42%
osmotic diuretics
42%
Spironolactone
42%
What are the antiarrhythmics? Class I
37%
Class II
37%
Class III
37%
Class IV
37%
What are the clinical indications for vasodilators? heart failure
37%
Nitroglycerin
37%
Diltiazem
32%
Diltiazem
32%
glaucoma
32%
hypertension
32%
What is an example of each? Lignocaine
32%
Propranolol
32%
Propranolol
32%
Sotalol
32%
aldosterone antagonist (aldosterone drives the Na+-K+- ATPase pump)
26%
Prazosin
26%
What are the common uses? to improve myocardial contractility
26%
β1 antagonism leading to decreased heart rate and negative inotropy, leading to decrease blood pressure
21%
What are they used for? fluid accumulation in the lungs, abdomen, or pleural space after heart failure
21%
Nitroprusside
21%
to prevent arrhythmias
21%
used as a prodrug that converts to nitric oxide, leading to vasodilation & reduced preload
21%
Angiotensin II receptor inhibitors
16%
increases plasma osmolality so moves fluid from tissues to plasma through osmosis
16%
used to improve myocardial contraction force to improve cardiac output
16%
blockade of sympathetic autonomic effects
11%
What will the use of vasodilators do? decrease afterload
11%
improve peripheral perfusion
11%
What is the mechanism of action of each? Na+-K+-2Cl- pump antagonist, therefore creating an enormous natriuresis (sodium in urine) and subsequent diuresis
11%
acts as an antagonist at the Angiotensin II receptor, therefore inhibiting the production of ADH and Aldosterone
5%
blocks the influx of calcium into cardiac and vascular smooth muscle cells, therefore decreasing myocardial contractility and causing vasodilation
5%
Class II antiarrhythmics
5%
Class II antiarrythmics
5%
What is the mechanism of action for each? inhibits the Angiotensin Converting Enzyme, therefore inhibiting the production of Angiotensin II
5%
What kind of agonists/antagonists are they? Na+ - K+ - ATPase channel antagonist
5%
Na+ - K+ - ATPase channel blocker
5%
β1 agonist
0%
α1 antagonists
0%
α1 selective antagonist, leading to vasodilation
0%
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